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F11

coagulation factor XI

The F11 gene provides instructions for making coagulation factor XI, a protein crucial for normal blood clot formation following injury. F11 encodes factor XI, a key component of the coagulation cascade that helps stop bleeding.

Chromosome 4q35.2 Autosomal recessive HGNC:3529 Tier C
F11 4q35.2 p arm q arm 4

F11 is located on the long (q) arm of chromosome 4, at band 4q35.2. Arm ratio per GRCh38 - banding schematic.

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Overview

The F11 gene is responsible for producing coagulation factor XI, a protein that plays a vital role in the body's haemostasis system. This system is a complex network of processes designed to stop bleeding and aid in wound healing. Factor XI contributes to the formation of stable blood clots, which are essential for sealing damaged blood vessels after an injury.

Disruptions to the F11 gene can lead to reduced or non-functional factor XI, impacting the body's ability to form clots effectively. This can result in an increased risk of bleeding, characteristic of conditions like Factor XI deficiency.

What the gene does

The F11 gene provides the blueprint for coagulation factor XI, a protein synthesised primarily by liver cells. Factor XI circulates in the bloodstream in an inactive state until it is activated as part of the coagulation cascade, a series of biochemical reactions that culminate in blood clot formation. Upon vascular injury, the cascade initiates, activating factor XI.

Once activated, factor XI interacts with other coagulation factors, contributing to the conversion of prothrombin into its active form, thrombin. Thrombin, in turn, is critical for transforming fibrinogen into fibrin. Fibrin strands then polymerise to form the mesh-like structure that stabilises a blood clot, effectively sealing the damaged blood vessel and halting blood loss.

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Chromosome location

The F11 gene is located on the long arm (q) of chromosome 4, specifically at position 4q35.2. This location indicates its precise address within the human genome. Understanding a gene's chromosomal location can be important for genetic mapping and identifying its proximity to other genes.

Protein structure

The coagulation factor XI protein, encoded by the F11 gene, consists of 625 amino acids. It features a distinct domain organisation essential for its function. The protein contains four Apple domains: Apple 1 (amino acids 20-103), Apple 2 (amino acids 110-193), Apple 3 (amino acids 200-283), and Apple 4 (amino acids 291-374). These are followed by a Peptidase S1 domain (amino acids 388-623), which is characteristic of serine proteases and is crucial for the protein's enzymatic activity in the coagulation cascade.

Domain map · 625 amino acids
Apple 1 (20–103)Apple 2 (110–193)Apple 3 (200–283)Apple 4 (291–374)Peptidase S1 (388–623)Apple 120–103Apple 2110–193Peptidase S1388–6231~313625
Domain - independent functional unit
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UniProt:P03951Length:625 aaStructure:AlphaFold

Key variants

Genetic variations within the F11 gene can alter the production or function of coagulation factor XI. These variations, often called mutations, can range from single nucleotide changes to larger deletions or insertions within the gene sequence. Over 250 different variants in the F11 gene have been identified that are associated with inherited bleeding conditions.

The table below shows the top 10 pathogenic or likely-pathogenic variants currently classified in ClinVar for F11.
View all on ClinVar →

Sample of pathogenic variants

10 pathogenic / likely-pathogenic variants from ClinVar, ranked by review status (expert-panel-reviewed first). This is a sample; recurrent founder variants in a specific population may not appear here - see the full ClinVar listing via the link above.

Variant (HGVS) Protein change Classification Evidence Associated condition
c.1026G>T
single nucleotide variant
p.Gly342= Pathogenic/Likely pathogenic ★★☆☆ Plasma factor XI deficiency
c.1060G>A
single nucleotide variant
p.Gly354Arg Pathogenic/Likely pathogenic ★★☆☆ Plasma factor XI deficiency
c.1103G>A
single nucleotide variant
p.Gly368Glu Pathogenic ★★☆☆ Hereditary factor XI deficiency disease
c.1443del
Deletion
p.Ile481fs Pathogenic ★★☆☆ Plasma factor XI deficiency
c.214C>T
single nucleotide variant
p.Arg72Ter Pathogenic ★★☆☆ Hereditary factor XI deficiency disease
c.325+1G>A
single nucleotide variant
- Pathogenic ★★☆☆ Plasma factor XI deficiency
c.569T>C
single nucleotide variant
p.Leu190Pro Pathogenic/Likely pathogenic ★★☆☆ Plasma factor XI deficiency
c.688T>A
single nucleotide variant
p.Cys230Ser Pathogenic/Likely pathogenic ★★☆☆ Hereditary factor XI deficiency disease
c.738G>A
single nucleotide variant
p.Trp246Ter Pathogenic ★★☆☆ Plasma factor XI deficiency
c.981C>A
single nucleotide variant
p.Cys327Ter Pathogenic ★★☆☆ Plasma factor XI deficiency

Evidence stars indicate ClinVar review status. Individual variant interpretation should always be performed by a qualified clinical laboratory - many variants remain classified as Variants of Uncertain Significance (VUS) pending more research.

Associated conditions

Pathogenic variants in the F11 gene are primarily associated with Factor XI deficiency, also known as Rosenthal syndrome. This condition is an inherited bleeding disorder characterised by reduced levels or activity of coagulation factor XI. While often mild, individuals with Factor XI deficiency may experience symptoms such as easy bruising, nosebleeds, prolonged bleeding after surgery or trauma, and bleeding into the skin or gums.

Inheritance pattern

Conditions caused by pathogenic F11 variants typically follow autosomal recessive inheritance.

Carrier parent 1 altered copy Carrier parent 1 altered copy Affected Carrier Carrier Unaffected Affected Carrier Unaffected Circles = females · Squares = males

When both parents are carriers, each child has a 25% chance of being affected, 50% of being a carrier, and 25% of being unaffected.

Carrier frequency by population How common is heterozygous F11 carrier status across ancestry groups?

UK clinical status

The F11 gene is recognised within the NHS Genomic Medicine Service due to its clinical significance. It is included on several NHS national genomic test directories. Specifically, F11 is part of the 'Bleeding and platelet disorders' (R90), 'Factor XI deficiency' (R120), and 'Inherited bleeding disorders' gene panels, all of which are classified as 'green' indicating strong evidence for gene-disease association and clinical utility.

Frequently asked questions

What is the F11 gene responsible for?

The F11 gene provides the genetic instructions for creating coagulation factor XI, a protein made primarily in the liver. This protein is a critical component of the body's blood clotting system, helping to form stable clots to stop bleeding after an injury.

What is Factor XI deficiency?

Factor XI deficiency is an inherited bleeding disorder caused by variants in the F11 gene. It results in lower levels or reduced activity of coagulation factor XI, which can lead to symptoms like easy bruising, prolonged bleeding, and nosebleeds.

How is Factor XI deficiency inherited?

Factor XI deficiency is typically inherited in an autosomal recessive pattern. This means an individual must inherit two altered copies of the F11 gene (one from each parent) to develop the condition. Individuals with one altered copy are usually carriers and do not typically show symptoms.

References

  1. Puy C, Rigg RA, McCarty OJ. The hemostatic role of factor XI. Thrombosis research. 2016. PMID: 27207433
  2. Peretz H, Salomon O, Mor-Cohen R. Type I mutation in the F11 gene is a third ancestral mutation which causes factor XI deficiency in Ashkenazi Jews. Journal of thrombosis and haemostasis : JTH. 2013. PMID: 23332144
  3. Duga S, Salomon O. Congenital factor XI deficiency: an update. Seminars in thrombosis and hemostasis. 2013. PMID: 23929304
  4. He R, Chen D, He S. Factor XI: hemostasis, thrombosis, and antithrombosis. Thrombosis research. 2012. PMID: 22197449
  5. Emsley J, McEwan PA, Gailani D. Structure and function of factor XI. Blood. 2010. PMID: 20110423
  6. Duga S, Salomon O. Factor XI Deficiency. Seminars in thrombosis and hemostasis. 2009. PMID: 19598070
  7. Gomez K, Bolton-Maggs P. Factor XI deficiency. Haemophilia : the official journal of the World Federation of Hemophilia. 2008. PMID: 18312365
Educational content. This page is not medical or genetic advice, is not individually reviewed by a clinician for each reader, and should not replace a consultation with a qualified healthcare professional or genetic counsellor. If you are considering genetic testing or acting on a test result, book a consultation.
Data sources Last updated 13 September 2026. Content compiled from HGNC · MedlinePlus Genetics · ClinGen · Genomics England PanelApp · NHS National Genomic Test Directory · ClinVar · UniProt · AlphaFold .